Voltage-Gated Channels as Causative Agents for Epilepsies
Mutasem Abuhamed1, Xiao Bo1, Kun Xia1, Yi Fang1 & LiLi Long1 Show affiliations
- Central South University, China
American Journal of Immunology, Volume 4, Issue 4, pp. 43–50 · doi.org/10.3844/ajisp.2008.43.50
Abstract
Problem statement: Epilepsy is a common neurological disorder that afflicts 1-2% of the general population worldwide. It encompasses a variety of disorders with seizures. Approach: Idiopathic epilepsies were defined as a heterogeneous group of seizure disorders that show no underlying cause .Voltage-gated ion channels defect were recognized etiology of epilepsy in the central nervous system. The aim of this article was to provide an update on voltage-gated channels and their mutation as causative agents for epilepsies. We described the structures of the voltage-gated channels, discuss their current genetic studies, and then review the effects of voltage-gated channels as causative agents for epilepsies. Results: Channels control the flow of ions in and out of the cell causing depolarization and hyper polarization of the cell. Voltage-gated channels were classified into four types: Sodium, potassium calcium ands chloride. Voltage-gated channels were macromolecular protein complexes within the lipid membrane. They were divided into subunits. Each subunit had a specific function and was encoded by more than one gen. Conclusion: Current genetic studies of idiopathic epilepsies show the importance of genetic influence on Voltage-gated channels. Different genes may regulate a function in a channel; the channel defect was directly responsible for neuronal hyper excitability and seizures.
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Abuhamed, M., Bo, X., Xia, K., Fang, Y. & Long, L. (2008). Voltage-Gated Channels as Causative Agents for Epilepsies. American Journal of Immunology, 4(4), 43–50. https://doi.org/10.3844/ajisp.2008.43.50
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