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<title>Volume 5, Issue 3 (2009) | American Journal of Immunology</title>
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    <title><![CDATA[Hyper-Ag-specific Ab Production in NC/Nga Mice is not associated with Deletion Polymorphism in the Promotor Region of the FcγRIIB Gene]]></title>
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        <![CDATA[<p>American Journal of Immunology, Published online: 31 December 2009; <a href="https://thescipub.com/aji/ajisp.2009.61.64">doi:10.3844/ajisp.2009.61.64</a></p>Problem statement: NC/Nga (NC) mice produced high levels of ovalbumin (OVA)-specific IgG, IgG1 and IgG2a. We previously found deletion polymorphisms in the promoter region of fcgr2b in NC mice. To det...]]></content:encoded>
    <dc:title><![CDATA[Hyper-Ag-specific Ab Production in NC/Nga Mice is not associated with Deletion Polymorphism in the Promotor Region of the FcγRIIB Gene]]></dc:title><dc:creator>Tohru  Sakai</dc:creator><dc:creator>Mariko  Nakamoto</dc:creator><dc:creator>Emi  Suto</dc:creator><dc:identifier>doi:10.3844/ajisp.2009.61.64</dc:identifier>
    <dc:source>American Journal of Immunology, Published online: 2009-12-31; | doi:10.3844/ajisp.2009.61.64</dc:source>
    <dc:date>2009-12-31</dc:date>
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    <title><![CDATA[Cytokines as Adjuvants for Vaccine and Cellular Therapies for Cancer]]></title>
    <link>https://thescipub.com/aji/ajisp.2009.65.83</link>
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        <![CDATA[<p>American Journal of Immunology, Published online: 30 September 2009; <a href="https://thescipub.com/aji/ajisp.2009.65.83">doi:10.3844/ajisp.2009.65.83</a></p>Problem statement: The development of a potent vaccine that can help treat tumors resistant to conventional cytotoxic therapies remains elusive. While part of the problem may be that trials have focus...]]></content:encoded>
    <dc:title><![CDATA[Cytokines as Adjuvants for Vaccine and Cellular Therapies for Cancer]]></dc:title><dc:creator>Christian M. Capitini</dc:creator><dc:creator>Terry J. Fry</dc:creator><dc:creator>Crystal L. Mackall</dc:creator><dc:identifier>doi:10.3844/ajisp.2009.65.83</dc:identifier>
    <dc:source>American Journal of Immunology, Published online: 2009-09-30; | doi:10.3844/ajisp.2009.65.83</dc:source>
    <dc:date>2009-09-30</dc:date>
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    <prism:doi>10.3844/ajisp.2009.65.83</prism:doi>
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    <title><![CDATA[Association of <i>Mycoplasma pneumoniae</i> Infection with Myocardial Infarction]]></title>
    <link>https://thescipub.com/aji/ajisp.2009.84.88</link>
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        <![CDATA[<p>American Journal of Immunology, Published online: 30 September 2009; <a href="https://thescipub.com/aji/ajisp.2009.84.88">doi:10.3844/ajisp.2009.84.88</a></p>Problem statement: In addition to the major risk factors for atherosclerosis such as high plasma level of low density lipoprotein, low plasma level of high density lipoprotein, cigarette smoking, hype...]]></content:encoded>
    <dc:title><![CDATA[Association of <i>Mycoplasma pneumoniae</i> Infection with Myocardial Infarction]]></dc:title><dc:creator>Morteza  Pourahmad</dc:creator><dc:creator>Abdolreza Sotoodeh Jahromy</dc:creator><dc:creator>Mohammad  Shojaei</dc:creator><dc:identifier>doi:10.3844/ajisp.2009.84.88</dc:identifier>
    <dc:source>American Journal of Immunology, Published online: 2009-09-30; | doi:10.3844/ajisp.2009.84.88</dc:source>
    <dc:date>2009-09-30</dc:date>
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    <prism:doi>10.3844/ajisp.2009.84.88</prism:doi>
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    <title><![CDATA[Mast Cells Inhibit CD8+ T Cell-Mediated Rejection of a Malignant Fibrous Histiocytoma-Like Tumor: Involvement of Fas-Fas Ligand Axis]]></title>
    <link>https://thescipub.com/aji/ajisp.2009.89.97</link>
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        <![CDATA[<p>American Journal of Immunology, Published online: 30 September 2009; <a href="https://thescipub.com/aji/ajisp.2009.89.97">doi:10.3844/ajisp.2009.89.97</a></p>Problem statement: Mast cells develop from bone marrow-derived progenitor cells and are distributed in the skin or mucosa where they play proinflammatory roles in the first line of defense.  Since som...]]></content:encoded>
    <dc:title><![CDATA[Mast Cells Inhibit CD8+ T Cell-Mediated Rejection of a Malignant Fibrous Histiocytoma-Like Tumor: Involvement of Fas-Fas Ligand Axis]]></dc:title><dc:creator>Hiroshi  Furukawa</dc:creator><dc:creator>Hiroshi  Kitazawa</dc:creator><dc:creator>Izumi  Kaneko</dc:creator><dc:creator>Koichi  Kikuchi</dc:creator><dc:creator>Shigeto  Tohma</dc:creator><dc:creator>Masato  Nose</dc:creator><dc:creator>Masao  Ono</dc:creator><dc:identifier>doi:10.3844/ajisp.2009.89.97</dc:identifier>
    <dc:source>American Journal of Immunology, Published online: 2009-09-30; | doi:10.3844/ajisp.2009.89.97</dc:source>
    <dc:date>2009-09-30</dc:date>
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    <title><![CDATA[Atopic Dermatitis and Type 1 Diabetes Mellitus in Iranian Children]]></title>
    <link>https://thescipub.com/aji/ajisp.2009.98.100</link>
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        <![CDATA[<p>American Journal of Immunology, Published online: 30 September 2009; <a href="https://thescipub.com/aji/ajisp.2009.98.100">doi:10.3844/ajisp.2009.98.100</a></p>Problem statement: Atopic diseases, including asthma, eczema and allergic rhinitis, are characterized by a chronic inflammatory reaction mediated by T helper 2 cells, while type 1 diabetes mellitus is...]]></content:encoded>
    <dc:title><![CDATA[Atopic Dermatitis and Type 1 Diabetes Mellitus in Iranian Children]]></dc:title><dc:creator>Elham  Ahmadi</dc:creator><dc:creator>Zahra  Rahnama</dc:creator><dc:creator>Ali Rikhtegaran Tehrani</dc:creator><dc:identifier>doi:10.3844/ajisp.2009.98.100</dc:identifier>
    <dc:source>American Journal of Immunology, Published online: 2009-09-30; | doi:10.3844/ajisp.2009.98.100</dc:source>
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