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<title>Volume 3, Issue 1 (2007) | American Journal of Immunology</title>
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    <title><![CDATA[HIV-1-Specific CTL Recognizing a Scarce Epitope Are Not Suppressed by HIV-1 Protease Inhibitors]]></title>
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        <![CDATA[<p>American Journal of Immunology, Published online: 30 June 2007; <a href="https://thescipub.com/aji/ajisp.2007.1.3">doi:10.3844/ajisp.2007.1.3</a></p>HIV-1-protease inhibitors have received growing attention as agents that can affect proteases in human proteasomes, with potentially deleterious effects for HIV-1-specific cellular immunity. Studying ...]]></content:encoded>
    <dc:title><![CDATA[HIV-1-Specific CTL Recognizing a Scarce Epitope Are Not Suppressed by HIV-1 Protease Inhibitors]]></dc:title><dc:creator>Otto O. Yang</dc:creator><dc:identifier>doi:10.3844/ajisp.2007.1.3</dc:identifier>
    <dc:source>American Journal of Immunology, Published online: 2007-06-30; | doi:10.3844/ajisp.2007.1.3</dc:source>
    <dc:date>2007-06-30</dc:date>
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    <title><![CDATA[Protection against Lipopolysaccharide-induced Death by An Anti-Interleukin-6 Monoclonal Antibody]]></title>
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        <![CDATA[<p>American Journal of Immunology, Published online: 30 June 2007; <a href="https://thescipub.com/aji/ajisp.2007.4.9">doi:10.3844/ajisp.2007.4.9</a></p>Septicemia is frequently associated with serious complications and high mortality despite recent advances in treatment with antibacterial agents. Infectious organisms or their soluble products initiat...]]></content:encoded>
    <dc:title><![CDATA[Protection against Lipopolysaccharide-induced Death by An Anti-Interleukin-6 Monoclonal Antibody]]></dc:title><dc:creator>Bailin  Liang</dc:creator><dc:creator>Debra  Gardner</dc:creator><dc:creator>Don  Griswold</dc:creator><dc:creator>Xiao-yu R. Song</dc:creator><dc:identifier>doi:10.3844/ajisp.2007.4.9</dc:identifier>
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    <title><![CDATA[The Pathology of Alzheimer Disease Elicits an In Vivo Immunological Response]]></title>
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        <![CDATA[<p>American Journal of Immunology, Published online: 30 June 2007; <a href="https://thescipub.com/aji/ajisp.2007.10.14">doi:10.3844/ajisp.2007.10.14</a></p>The mechanism(s) responsible for the loss of neurons that characterizes Alzheimer disease is incompletely understood. Nonetheless, there is considerable evidence suggestive of immune abnormalities cou...]]></content:encoded>
    <dc:title><![CDATA[The Pathology of Alzheimer Disease Elicits an In Vivo Immunological Response]]></dc:title><dc:creator>David J. Bonda</dc:creator><dc:creator>Kate M. Webber</dc:creator><dc:creator>Sandra L. Siedlak</dc:creator><dc:creator>George  Perry</dc:creator><dc:creator>Robert P. Friedland</dc:creator><dc:creator>Mark A. Smith</dc:creator><dc:identifier>doi:10.3844/ajisp.2007.10.14</dc:identifier>
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    <title><![CDATA[Expression of Toll-like Receptors (TLRs) and Nucleotide-binding Oligomerization Domain (NODs) in murine peritoneal macrophages on <i>in vitro</i> treatment with Thymosin Î± 1]]></title>
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        <![CDATA[<p>American Journal of Immunology, Published online: 30 June 2007; <a href="https://thescipub.com/aji/ajisp.2007.15.24">doi:10.3844/ajisp.2007.15.24</a></p>Thymosin α 1 (Tα1) treatment of murine peritoneal macrophages in vitro induces the expression of TLRs and Nod2 proteins. Enhanced expressions of TLR2, -4, -5, -6, -7, -8, -9 and Nod2 were observed b...]]></content:encoded>
    <dc:title><![CDATA[Expression of Toll-like Receptors (TLRs) and Nucleotide-binding Oligomerization Domain (NODs) in murine peritoneal macrophages on <i>in vitro</i> treatment with Thymosin Î± 1]]></dc:title><dc:creator>Shikha  Tarang</dc:creator><dc:creator>Ajit  Sodhi</dc:creator><dc:identifier>doi:10.3844/ajisp.2007.15.24</dc:identifier>
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